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Jeffrey (J) Kroon

Department
Molecular Cell Biology
E-mail
j.kroon@sanquin.nl
LinkedIn
http://nl.linkedin.com/pub/jeffrey-kroon/24/a2b/114
Training

Cell biology/Biochemistry

Research interests
Endothelial cells lining the lumen of arteries are constantly subjected to a high pulsatile laminar flow and high mechanical shear stress. This high shear stress plays an important role in vascular remodeling and has an atheroprotective function, whereas turbulent flow associated with low shear stress is related with the on-set of diseases such as atherosclerosis. Therefore, it is important to understand how endothelial cells sense the changes in physiological shear stress and adapt in response to flow. A key-event in response to flow is re-arrangement of the actin cytoskeleton. It is known that Rho GTPases play a pivotal role in this process. Our current focus is identifying the pathways involved in Rho GTPase activation and its upstream activators under physiological flow conditions.
Technology
  • In vitro flow assays
  • Pulldown and immunoprecipitation assays for analysis of protein-protein interactions
  • Confocal microscopy and live cell imaging
  • Cell culturing
  • Western blotting
Resume
2010-hedenPhD-student at the Dept of Molecular Cell Biology, Sanquin Research, Amsterdam
2010MSc in Biomolecular Sciences at the VU University, Amsterdam 
2010Internship at the Department of Genetics & Pathology: Vascular Biology, Uppsala University, Uppsala, Sweden
2009Internship at the department of Oral Microbiology, Academic Centre for Dentistry, Amsterdam
2008BSc in Biochemistry, INHolland, Alkmaar
2007-2008Internship at the Dept of Molecular Cell Biology, Sanquin Research, Amsterdam
2006Internship at the Dept of Molecular Cell Biology and Immunology, VU Medical Centre (VUmc), Amsterdam
Publications
Sanquin publications
Other publications

van der Bij GJ, Bögels M, Oosterling SJ, Kroon J, Schuckmann DT, de Vries HE, Meijer S, Beelen RH, van Egmond M. Tumor infiltrating macrophages reduce development of peritoneal colorectal carcinoma metastases. Cancer Lett 2008; 262(1):77-86.

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